Suppression of PTEN function increases breast cancer chemotherapeutic drug resistance while conferring sensitivity to mTOR inhibitors-Reference-Cited by-同舟云学术

Suppression of PTEN function increases breast cancer chemotherapeutic drug resistance while conferring sensitivity to mTOR inhibitors

Published:2008-03-10 Issue:29 Volume:27 Page:4086-4095
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Steelman L S,Navolanic P M,Sokolosky M L,Taylor J R,Lehmann B D,Chappell W H,Abrams S L,Wong E W T,Stadelman K M,Terrian D M,Leslie N R,Martelli A M,Stivala F,Libra M,Franklin R A,McCubrey J A

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc200849.pdf

Reference36 articles.

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2. Agrawal S, Eng C . (2006). Differential expression of novel naturally occurring splice variants of PTEN and their functional consequences in Cowden syndrome and sporadic breast cancer. Hu Mol Gen 5: 777–787.

3. Alessi DR, James SR, Downes CP, Holmes AB, Gaffney PR, Reese CB et al. (1997). Characterization of a 3-phosphoinositide-dependent protein kinase which phosphorylates and activates protein kinase Balpha. Curr Biol 7: 261–269.

4. Bose S, Crane A, Hibshoosh H, Mansukhani M, Sandweis L, Parsons R . (2002). Reduced expression of PTEN correlates with breast cancer progression. Hu Pathol 33: 405–409.

5. Chiang GG, Abraham RT . (2005). Phosphorylation of mammalian target of rapamycin (mTOR) at Ser-2448 is mediated by p70S6 kinase. J Biol Chem 280: 25485–25490.

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