High susceptibility to bacterial infection, but no liver dysfunction, in mice compromised for hepatocyte NF-κB activation-Reference-Cited by-同舟云学术

High susceptibility to bacterial infection, but no liver dysfunction, in mice compromised for hepatocyte NF-κB activation

Published:2000-05 Issue:5 Volume:6 Page:573-577
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Lavon Iris,Goldberg Iris,Amit Sharon,Landsman Limor,Jung Steffen,Tsuberi Ben-Zion,Barshack Iris,Kopolovic Juri,Galun Eitan,Bujard Hermann,Ben-Neriah Yinon

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm0500_573.pdf

Reference25 articles.

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3. Beg, A.A., Sha, W.C., Bronson, R.T., Ghosh, S. & Baltimore, D. Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-κB. Nature 376, 167–169 (1995).

4. Li, Z.-W. et al. The IKKβ subunit of IκB kinase (IKK) is essential for NF-κB activation and prevention of apoptosis. J. Exp. Med. 189, 1–7 (1999).

5. Li, Q., Van Antwerp, D., Mercurio, F., Lee, K.-F. & Verma, I.M. Severe liver degeneration in mice lacking the IκB kinase 2 gene. Science 284, 321–325 (1999).

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