IκBζ controls IL-17-triggered gene expression program in intestinal epithelial cells that restricts colonization of SFB and prevents Th17-associated pathologies
Author:
Publisher
Elsevier BV
Subject
Immunology,Immunology and Allergy
Link
https://www.nature.com/articles/s41385-022-00554-3.pdf
Reference75 articles.
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2. Gut microbiota: role in pathogen colonization, immune responses, and inflammatory disease;Pickard;Immunol. Rev.,2017
3. Guardians of the Gut: Enteric Defensins;Sankaran-Walters;Front. Microbiol.,2017
4. Cytokine Tuning of Intestinal Epithelial Function;Andrews;Front. Immunol.,2018
5. Interaction Between the Microbiota, Epithelia, and Immune Cells in the Intestine;Kayama;Annu. Rev. Immunol.,2020
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1. The Nuclear NF-κB Regulator IκBζ: Updates on Its Molecular Functions and Pathophysiological Roles;Cells;2024-08-31
2. IκBζ is an essential mediator of immunity to oropharyngeal candidiasis;Cell Host & Microbe;2023-10
3. The central inflammatory regulator IκBζ: induction, regulation and physiological functions;Frontiers in Immunology;2023-06-12
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