Hyper-active non-homologous end joining selects for synthetic lethality resistant and pathological Fanconi anemia hematopoietic stem and progenitor cells-Reference-Cited by-同舟云学术

Hyper-active non-homologous end joining selects for synthetic lethality resistant and pathological Fanconi anemia hematopoietic stem and progenitor cells

Published:2016-02-26 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Du Wei,Amarachintha Surya,Wilson Andrew F.,Pang Qishen

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep22167.pdf

Reference61 articles.

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2. Tischkowitz, M. D. & Hodgson, S. V. Fanconi anaemia. J Med Genet 40, 1–10 (2003).

3. Kennedy, R. D. & D’Andrea, A. D. The Fanconi anemia/BRCA pathway: new faces in the crowd. Genes Dev. 19, 2925–2940 (2000).

4. Timmers, C. et al. Positional cloning of a novel Fanconi anemia gene, FANCD2. Mol Cell 7(2), 241–248 (2001).

5. Bogliolo, M. et al. Mutations in ERCC4, encoding the DNA-repair endonuclease XPF, cause Fanconi anemia. Am. J. Hum. Genet. 92(5), 800–806 (2013).

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