Immune privilege or inflammation? Insights into the Fas ligand enigma-Reference-Cited by-同舟云学术

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Immune privilege or inflammation? Insights into the Fas ligand enigma

Published:2001-03 Issue:3 Volume:7 Page:271-274
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

O'Connell Joe,Houston Aileen,Bennett Michael W.,O'Sullivan Gerald C.,Shanahan Fergus

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm0301_271.pdf

Reference40 articles.

1. Restifo, N.P. Not so Fas: re-evaluating the mechanisms of immune privilege and tumor escape. Nature Med. 6, 493–495 (2000).

2. Griffith, T.S. & Ferguson, T.A. The role of FasL-induced apoptosis in immune privilege. Immunol. Today 18, 240–244 (1997).

3. Lepple-Wienhues, A. et al. Stimulation of CD95 (Fas) blocks T lymphocyte calcium channels through sphingomyelinase and sphingolipids. Proc. Natl. Acad. Sci. USA 96, 13795–13800 (1999).

4. Sampalo, A. et al. Chronic lymphocytic leukemia B cells inhibit spontaneous Ig production by autologous bone marrow cells: role of CD95-CD95L interaction. Blood 96, 3168–3174 (2000).

5. Kang, S.-M. et al. Fas ligand expression in islets of Langerhans does not confer immune privilege and instead targets them for rapid destruction. Nature Med. 3, 738–743 (1997).

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