Ribosome stalling during selenoprotein translation exposes a ferroptosis vulnerability-Reference-Cited by-同舟云学术

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Ribosome stalling during selenoprotein translation exposes a ferroptosis vulnerability

Published:2022-05-30 Issue:7 Volume:18 Page:751-761
ISSN:1552-4450
Container-title:Nature Chemical Biology
language:en
Short-container-title:Nat Chem Biol

Author:

Li Zhipeng^ORCID,Ferguson Lucas,Deol Kirandeep K.,Roberts Melissa A.,Magtanong Leslie^ORCID,Hendricks Joseph M.,Mousa Gergey Alzaem,Kilinc Seda,Schaefer Kaitlin,Wells James A.^ORCID,Bassik Michael C.^ORCID,Goga Andrei^ORCID,Dixon Scott J.^ORCID,Ingolia Nicholas T.^ORCID,Olzmann James A.^ORCID

Funder

American Cancer Society

Melanoma Research Alliance

U.S. Department of Health & Human Services | National Institutes of Health

Chan Zuckerberg Biohub, Miller Institute

United States Department of Defense | United States Army | Army Medical Command | Congressionally Directed Medical Research Programs

Mark Foundation

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

https://www.nature.com/articles/s41589-022-01033-3.pdf

Reference60 articles.

1. Dixon, S. J. et al. Ferroptosis: an iron-dependent form of nonapoptotic cell death. Cell 149, 1060–1072 (2012).

2. Jiang, X., Stockwell, B. R. & Conrad, M. Ferroptosis: mechanisms, biology and role in disease. Nat. Rev. Mol. Cell Biol. 22, 266–282 (2021).

3. Yang, W. S. et al. Regulation of ferroptotic cancer cell death by GPX4. Cell 156, 317–331 (2014).

4. Bersuker, K. et al. The CoQ oxidoreductase FSP1 acts parallel to GPX4 to inhibit ferroptosis. Nature 575, 688–692 (2019).

5. Doll, S. et al. FSP1 is a glutathione-independent ferroptosis suppressor. Nature 575, 693–698 (2019).

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