GAD1 (2q31.1), which encodes glutamic acid decarboxylase (GAD67), is associated with childhood-onset schizophrenia and cortical gray matter volume loss-Reference-Cited by-同舟云学术

GAD1 (2q31.1), which encodes glutamic acid decarboxylase (GAD67), is associated with childhood-onset schizophrenia and cortical gray matter volume loss

Published:2004-10-26 Issue:6 Volume:10 Page:581-588
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Addington A M,Gornick M,Duckworth J,Sporn A,Gogtay N,Bobb A,Greenstein D,Lenane M,Gochman P,Baker N,Balkissoon R,Vakkalanka R K,Weinberger D R,Rapoport J L,Straub R E

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/4001599.pdf

Reference43 articles.

1. Harrison PJ, Weinberger DR . Schizophrenia genes, gene expression, and neuropathology: on the matter of their convergence. Mol Psychiatry 2004.

2. Blum BP, Mann JJ . The GABAergic system in schizophrenia. Int J Neuropsychopharmacol 2002; 5: 159–179.

3. Kalkman HO, Loetscher E . GAD(67): the link between the GABA-deficit hypothesis and the dopaminergic- and glutamatergic theories of psychosis. J Neural Transm 2003; 110: 803–812.

4. Bu DF, Tobin AJ . The exon–intron organization of the genes (GAD1 and GAD2) encoding two human glutamate decarboxylases (GAD67 and GAD65) suggests that they derive from a common ancestral GAD. Genomics 1994; 21: 222–228.

5. Soghomonian JJ, Martin DL . Two isoforms of glutamate decarboxylase: why? Trends Pharmacol Sci 1998; 19: 500–505.

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