Variants of the serotonin transporter gene (SLC6A4) significantly contribute to hyperserotonemia in autism-Reference-Cited by-同舟云学术

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Variants of the serotonin transporter gene (SLC6A4) significantly contribute to hyperserotonemia in autism

Published:2004-03 Issue:3 Volume:9 Page:264-271
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Coutinho A M,Oliveira G,Morgadinho T,Fesel C,Macedo T R,Bento C,Marques C,Ataíde A,Miguel T,Borges L,Vicente A M

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/4001409.pdf

Reference44 articles.

1. Piven J . The biological basis of autism. Curr Opin Neurobiol 1997; 7: 708–712.

2. Lamb JA, Moore J, Bailey A, Monaco AP . Autism: recent molecular genetic advances. Hum Mol Genet 2000; 9: 861–868.

3. Folstein SE, Rosen-Sheidley B . Genetics of autism: complex aetiology for a heterogeneous disorder. Nat Rev Genet 2001; 2: 943–955.

4. Newbury DF, Bonora E, Lamb JA, Fisher SE, Lai CSL, Baird G et al. FOXP2 is not a major susceptibility gene for autism or specific language impairment. Am J Hum Genet 2002; 70: 1318–1327.

5. Buxbaum JD, Silverman JM, Smith CJ, Kilifarski M, Reichert J, Hollander E et al. Evidence for a susceptibility gene for autism on chromosome 2 and for genetic heterogeneity. Am J Hum Genet 2001; 68: 1514–1520.

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