β2-Adrenergic receptor gene variants and risk for autism in the AGRE cohort-Reference-Cited by-同舟云学术

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β2-Adrenergic receptor gene variants and risk for autism in the AGRE cohort

Published:2007-01-02 Issue:3 Volume:12 Page:283-291
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Cheslack-Postava K,Fallin M D,Avramopoulos D,Connors S L,Zimmerman A W,Eberhart C G,Newschaffer C J

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/4001940.pdf

Reference62 articles.

1. Arndt TL, Stodgell CJ, Rodier PM . The teratology of autism. Int J Dev Neurosci 2005; 23: 189–199.

2. Weiss ER, Maness P, Lauder JM . Why do neurotransmitters act like growth factors? Perspect Dev Neurobiol 1998; 5: 323–335.

3. Schlumpf M, Bruinink A, Lichtensteiger W, Cortes R, Palacios JM, Pazos A . Beta-adrenergic binding sites in fetal rat central nervous system and pineal gland: their relation to other receptor sites. Dev Pharmacol Ther 1987; 10: 422–435.

4. Slotkin TA, Lau C, Seidler FJ . Beta-adrenergic receptor overexpression in the fetal rat: distribution, receptor subtypes, and coupling to adenylate cyclase activity via G-proteins. Toxicol Appl Pharmacol 1994; 129: 223–234.

5. Garofolo MC, Seidler FJ, Cousins MM, Tate CA, Qiao D, Slotkin TA . Developmental toxicity of terbutaline: critical periods for sex-selective effects on macromolecules and DNA synthesis in rat brain, heart, and liver. Brain Res Bull 2003; 59: 319–329.

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