A distinct bacterial dysbiosis associated skin inflammation in ovine footrot
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep45220.pdf
Reference73 articles.
1. Egerton, J. R., Roberts, D. S. & Parsonson, I. The Aetiology and Pathogenesis of ovine Footrot. J. comp. Pathol. 79, 207–217 (1969).
2. Wassink, G. J., George, T. R., Kaler, J. & Green, L. E. Footrot and interdigital dermatitis in sheep: farmer satisfaction with current management, their ideal management and sources used to adopt new strategies. Prev Vet Med 96, 65–73 (2010).
3. Kennan, R. M., Dhungyel, O. P., Whittington, R. J., Egerton, J. R. & Rood, J. I. The type IV fimbrial subunit gene (fimA) of Dichelobacter nodosus is essential for virulence, protease secretion, and natural competence. J Bacteriol 183, 4451–4458 (2001).
4. Kennan, R. M. et al. The subtilisin-like protease AprV2 is required for virulence and uses a novel disulphide-tethered exosite to bind substrates. PLoS Pathog 6, e1001210 (2010).
5. Witcomb, L. A. et al. A longitudinal study of the role of Dichelobacter nodosus and Fusobacterium necrophorum load in initiation and severity of footrot in sheep. Prev Vet Med 115, 48–55 (2014).
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