Brain-Specific Inactivation of the Crhr1 Gene Inhibits Post-Dependent and Stress-Induced Alcohol Intake, but Does Not Affect Relapse-Like Drinking-Reference-Cited by-同舟云学术

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Brain-Specific Inactivation of the Crhr1 Gene Inhibits Post-Dependent and Stress-Induced Alcohol Intake, but Does Not Affect Relapse-Like Drinking

Published:2011-11-23 Issue:4 Volume:37 Page:1047-1056
ISSN:0893-133X
Container-title:Neuropsychopharmacology
language:en
Short-container-title:Neuropsychopharmacol

Author:

Molander Anna,Vengeliene Valentina,Heilig Markus,Wurst Wolfgang,Deussing Jan M,Spanagel Rainer

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Link

http://www.nature.com/articles/npp2011297.pdf

Reference55 articles.

1. Barr CS (2010). Commentary on Nelson et al (2010). Addict Biol 15: 12–14.

2. Becker HC, Lopez MF (2004). Increased ethanol drinking after repeated chronic ethanol exposure and withdrawal experience in C57BL/6 mice. Alcohol Clin Exp Res 28: 1829–1838.

3. Blomeyer D, Treutlein J, Esser G, Schmidt MH, Schumann G, Laucht M (2008). Interaction between CRFR1 gene and stressful life events predicts adolescent heavy alcohol use. Biol Psychiatry 63: 146–151.

4. Chu K, Koob GF, Cole M, Zorrilla EP, Roberts AJ (2007). Dependence-induced increases in ethanol self-administration in mice are blocked by the CRF1 receptor antagonist antalarmin and by CRF1 receptor knockout. Pharmacol Biochem Behav 86: 813–821.

5. Contet C, Gardon O, Filliol D, Becker JA, Koob GF, Kieffer BL (2011). Identification of genes regulated in the mouse extended amygdala by excessive ethanol drinking associated with dependence. Addict Biol 16: 615–619.

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