The innate immune sensor NLRC3 attenuates Toll-like receptor signaling via modification of the signaling adaptor TRAF6 and transcription factor NF-κB-Reference-Cited by-同舟云学术

The innate immune sensor NLRC3 attenuates Toll-like receptor signaling via modification of the signaling adaptor TRAF6 and transcription factor NF-κB

Published:2012-08-05 Issue:9 Volume:13 Page:823-831
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Schneider Monika,Zimmermann Albert G,Roberts Reid A,Zhang Lu,Swanson Karen V,Wen Haitao,Davis Beckley K,Allen Irving C,Holl Eda K,Ye Zhengmao,Rahman Adeeb H,Conti Brian J,Eitas Timothy K,Koller Beverly H,Ting Jenny P-Y

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni.2378.pdf

Reference50 articles.

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3. Lamkanfi, M. & Dixit, V.M. Modulation of inflammasome pathways by bacterial and viral pathogens. J. Immunol. 187, 597–602 (2011).

4. Davis, B.K. et al. Cutting Edge: NLRC5-dependent activation of the inflammasome. J. Immunol. 186, 1333–1337 (2011).

5. Bruey, J.M. et al. PAN1/NALP2/PYPAF2, an inducible inflammatory mediator that regulates NF-κB and caspase-1 activation in macrophages. J. Biol. Chem. 279, 51897–51907 (2004).

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