Telomeres avoid end detection by severing the checkpoint signal transduction pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/nature09353.pdf
Reference33 articles.
1. de Lange, T. How telomeres solve the end-protection problem. Science 326, 948–952 (2009)
2. Li, B., Oestreich, S. & de Lange, T. Identification of human Rap1: implications for telomere evolution. Cell 101, 471–483 (2000)
3. Du, L.-L., Nakamura, T. M. & Russell, P. Histone modification-dependent and -independent pathways for recruitment of checkpoint protein Crb2 to double-strand breaks. Genes Dev. 20, 1583–1596 (2006)
4. Greeson, N. T., Sengupta, R., Arida, A. R., Jenuwein, T. & Sanders, S. L. Di-methyl H4 lysine 20 targets the checkpoint protein Crb2 to sites of DNA damage. J. Biol. Chem. 283, 33168–33174 (2008)
5. Nakamura, T. M., Du, L.-L., Redon, C. & Russell, P. Histone H2A phosphorylation controls Crb2 recruitment at DNA breaks, maintains checkpoint arrest, and influences DNA repair in fission yeast. Mol. Cell. Biol. 24, 6215–6230 (2004)
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