Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1-Reference-Cited by-同舟云学术

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Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1

Published:2010-11-10 Issue:7324 Volume:468 Page:696-700
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Guzman Jaime N.,Sanchez-Padilla Javier,Wokosin David,Kondapalli Jyothisri,Ilijic Ema,Schumacker Paul T.,Surmeier D. James

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature09536.pdf

Reference33 articles.

1. Albin, R. L., Young, A. B. & Penney, J. B. The functional anatomy of disorders of the basal ganglia. Trends Neurosci. 18, 63–64 (1995)

2. Schapira, A. H. Mitochondria in the aetiology and pathogenesis of Parkinson’s disease. Lancet Neurol. 7, 97–109 (2008)

3. Puopolo, M., Raviola, E. & Bean, B. P. Roles of subthreshold calcium current and sodium current in spontaneous firing of mouse midbrain dopamine neurons. J. Neurosci. 27, 645–656 (2007)

4. Chan, C. S. et al. ‘Rejuvenation’ protects neurons in mouse models of Parkinson’s disease. Nature 447, 1081–1086 (2007)

5. Khaliq, Z. M. & Bean, B. P. Pacemaking in dopaminergic ventral tegmental area neurons: depolarizing drive from background and voltage-dependent sodium conductances. J. Neurosci. 30, 7401–7413 (2010)

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