Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7-Reference-Cited by-同舟云学术

Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7

Published:2015-12 Issue:1 Volume:6 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Minett Michael S.,Pereira Vanessa,Sikandar Shafaq,Matsuyama Ayako,Lolignier Stéphane,Kanellopoulos Alexandros H.,Mancini Flavia,Iannetti Gian D.,Bogdanov Yury D.,Santana-Varela Sonia,Millet Queensta,Baskozos Giorgios,MacAllister Raymond,Cox James J.,Zhao Jing,Wood John N.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms9967.pdf

Reference38 articles.

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2. Eijkelkamp, N. et al. Neurological perspectives on voltage-gated sodium channels. Brain 135, 2585–2612 (2012).

3. Waxman, S. G. et al. Sodium channel genes in pain-related disorders: phenotype-genotype associations and recommendations for clinical use. Lancet Neurol. 13, 1152–1160 (2014).

4. Zhang, Q. et al. Na+ current properties in islet alpha- and beta-cells reflect cell-specific Scn3a and Scn9a expression. J. Physiol. 592, 4677–4696 (2014).

5. Weiss, J. et al. Loss-of-function mutations in sodium channel Nav1.7 cause anosmia. Nature 472, 186–190 (2011).

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