Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway-Reference-Cited by-同舟云学术

Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway

Published:2003-12-14 Issue:1 Volume:10 Page:80-86
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Yasoda Akihiro,Komatsu Yasato,Chusho Hideki,Miyazawa Takashi,Ozasa Ami,Miura Masako,Kurihara Tatsuya,Rogi Tomohiro,Tanaka Shoji,Suda Michio,Tamura Naohisa,Ogawa Yoshihiro,Nakao Kazuwa

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm971.pdf

Reference44 articles.

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2. Bellus, G.A. et al. Achondroplasia is defined by recurrent G380R mutations of FGFR3. Am. J. Hum. Genet. 56, 368–373 (1995).

3. Shiang, R. et al. Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia. Cell 78, 335–342 (1994).

4. Cohen, M.M. Jr. Short-limb skeletal dysplasias and craniosynostosis: what do they have in common? Pediatr. Radiol. 27, 442–446 (1997).

5. Boilly, B., Vercoutter-Edouart, A.S., Hondermarck, H., Nurcombe, V. & Le Bourhis, X. FGF signals for cell proliferation and migration through different pathways. Cytokine Growth Factor Rev. 11, 295–302 (2000).

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