Brain PPAR-γ promotes obesity and is required for the insulin–sensitizing effect of thiazolidinediones-Reference-Cited by-同舟云学术

Brain PPAR-γ promotes obesity and is required for the insulin–sensitizing effect of thiazolidinediones

Published:2011-05 Issue:5 Volume:17 Page:618-622
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Lu Min,Sarruf David A,Talukdar Saswata,Sharma Shweta,Li Pingping,Bandyopadhyay Gautam,Nalbandian Sarah,Fan WuQiang,Gayen Jiaur R,Mahata Sushil K,Webster Nicholas J,Schwartz Michael W,Olefsky Jerrold M

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm.2332.pdf

Reference19 articles.

1. He, W. et al. Adipose-specific peroxisome proliferator–activated receptor γ knockout causes insulin resistance in fat and liver but not in muscle. Proc. Natl. Acad. Sci. USA 100, 15712–15717 (2003).

2. Hevener, A.L. et al. Muscle-specific PPAR-γ deletion causes insulin resistance. Nat. Med. 9, 1491–1497 (2003).

3. Hevener, A.L. et al. Macrophage PPAR-γ is required for normal skeletal muscle and hepatic insulin sensitivity and full antidiabetic effects of thiazolidinediones. J. Clin. Invest. 117, 1658–1669 (2007).

4. Odegaard, J.I. et al. Macrophage-specific PPAR-γ controls alternative activation and improves insulin resistance. Nature 447, 1116–1120 (2007).

5. Sarruf, D.A. et al. Expression of peroxisome proliferator–activated receptor-γ in key neuronal subsets regulating glucose metabolism and energy homeostasis. Endocrinology 150, 707–712 (2009).

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