Differential signaling networks of Bcr–Abl p210 and p190 kinases in leukemia cells defined by functional proteomics-Reference-Cited by-同舟云学术

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Differential signaling networks of Bcr–Abl p210 and p190 kinases in leukemia cells defined by functional proteomics

Published:2017-01-23 Issue:7 Volume:31 Page:1502-1512
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Reckel S,Hamelin R,Georgeon S,Armand F,Jolliet Q,Chiappe D,Moniatte M,Hantschel O

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu201736.pdf

Reference44 articles.

1. O'Hare T, Zabriskie MS, Eiring AM, Deininger MW . Pushing the limits of targeted therapy in chronic myeloid leukaemia. Nat Rev Cancer 2012; 12: 513–526.

2. Wong S, Witte ON . The BCR-ABL story: bench to bedside and back. Annu Rev Immunol 2004; 22: 247–306.

3. Deininger MW, Goldman JM, Melo JV . The molecular biology of chronic myeloid leukemia. Blood 2000; 96: 3343–3356.

4. Hantschel O . Structure, regulation, signaling, and targeting of abl kinases in cancer. Genes Cancer 2012; 3: 436–446.

5. Gambacorti-Passerini C, Antolini L, Mahon F-X, Guilhot F, Deininger M, Fava C et al. Multicenter independent assessment of outcomes in chronic myeloid leukemia patients treated with imatinib. J Natl Cancer Inst 2011; 103: 553–561.

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