Using the optimal method—explained variance weighted genetic risk score to predict the efficacy of folic acid therapy to hyperhomocysteinemia
Author:
Funder
Science and Technology Department of Henan Province
Publisher
Springer Science and Business Media LLC
Subject
Nutrition and Dietetics,Medicine (miscellaneous)
Link
https://www.nature.com/articles/s41430-021-01055-5.pdf
Reference40 articles.
1. Ohishi T, Fujita T, Suzuki D, Nishida T, Asukai M, Matsuyama Y. Serum homocysteine levels are affected by renal function during a 3-year period of minodronate therapy in female osteoporotic patients. J Bone Miner Metab. 2019;37:319–26.
2. Messedi M, Frigui M, Chaabouni K, Turki M, Neifer M, Lahiyani A, et al. Methylenetetrahydrofolate reductase C677T and A1298C polymorphisms and variations of homocysteine concentrations in patients with Behcet’s disease. Gene. 2013;527:306–10.
3. Peng HY, Man CF, Xu J, Fan Y. Elevated homocysteine levels and risk of cardiovascular and all-cause mortality: a meta-analysis of prospective studies. J Zhejiang Univ Sci B. 2015;16:78–86.
4. Schaffer A, Verdoia M, Cassetti E, Marino P, Suryapranata H, De Luca G, et al. Relationship between homocysteine and coronary artery disease. results from a large prospective cohort study. Thromb Res. 2014;134:288–93.
5. Cheng X. Updating the relationship between hyperhomocysteinemia lowering therapy and cardiovascular events. Cardiovasc Ther. 2013;31:e19–26.
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