High-throughput fitness screens link genes to unique phenotypes in human-restricted Salmonella
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Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41588-024-01780-0.pdf
Reference5 articles.
1. Wang, B. X., Butler, D. S., Hamblin, M. & Monack, D. M. One species, different diseases: the unique molecular mechanisms that underlie the pathogenesis of typhoidal Salmonella infections. Curr. Opin. Microbiol. 72, 102262 (2023). A review article that presents a summary of genetic differences between non-typhoidal and typhoidal Salmonella.
2. McClelland, M. et al. Comparison of genome degradation in Paratyphi A and Typhi, human-restricted serovars of Salmonella enterica that cause typhoid. Nat. Genet. 36, 1268–1274 (2004). This paper reports genomic comparisons between S. Typhiand S. Paratyphi A.
3. Holt, K. E. et al. High-throughput sequencing provides insights into genome variation and evolution in Salmonella Typhi. Nat. Genet. 40, 987–993 (2008). This paper uses bioinformatics to more deeply characterize the genomes of S. Typhi.
4. Wetmore, K. M. et al. Rapid quantification of mutant fitness in diverse bacteria by sequencing randomly bar-coded transposons. mBio 6, 1–15 (2015). This paper explains the principle and development of Rb-Tn-seq.
5. Leshchiner, D. et al. A genome-wide atlas of antibiotic susceptibility targets and pathways to tolerance. Nat. Commun. 13, 1–17 (2022). This paper applies SAFE to systematically study the fitness of a Gram-positive pathogen.
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