CCL22 mutations drive natural killer cell lymphoproliferative disease by deregulating microenvironmental crosstalk

Author:

Baer Constance,Kimura ShunsukeORCID,Rana Mitra S.ORCID,Kleist Andrew B.,Flerlage Tim,Feith David J.ORCID,Chockley PeterORCID,Walter WenckeORCID,Meggendorfer Manja,Olson Thomas L.,Cheon HeeJin,Olson Kristine C.,Ratan AakroshORCID,Mueller Martha-Lena,Foran James M.,Janke Laura J.,Qu Chunxu,Porter Shaina N.,Pruett-Miller Shondra M.ORCID,Kalathur Ravi C.ORCID,Haferlach Claudia,Kern Wolfgang,Paietta Elisabeth,Thomas Paul G.ORCID,Babu M. MadanORCID,Loughran Thomas P.,Iacobucci IlariaORCID,Haferlach TorstenORCID,Mullighan Charles G.ORCID

Funder

Center for Strategic Scientific Initiatives, National Cancer Institute

Henry Schueler 41 & 9 Foundation St. Baldrick’s Foundation Robert J. Arceci Innovation Award

Garwood Postdoctoral Fellowship of the Hematological Malignancies Program of the St Jude Children’s Research Hospital Comprehensive Cancer Center

U.S. Department of Health & Human Services | NIH | National Institute of General Medical Sciences

U.S. Department of Health & Human Services | NIH | U.S. National Library of Medicine

the Bess Family Charitable Fund, the LGL Leukemia Foundation and a generous anonymous donor

Publisher

Springer Science and Business Media LLC

Subject

Genetics

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