Cathepsin B S-nitrosylation promotes ADAR1-mediated editing of its own mRNA transcript via an ADD1/MATR3 regulatory axis-Reference-Cited by-同舟云学术

Cathepsin B S-nitrosylation promotes ADAR1-mediated editing of its own mRNA transcript via an ADD1/MATR3 regulatory axis

Published:2023-05-08 Issue:7 Volume:33 Page:546-561
ISSN:1748-7838
Container-title:Cell Research
language:en
Short-container-title:Cell Res

Author:

Lin Zhe,Zhao Shuang,Li Xuesong,Miao Zian,Cao Jiawei,Chen Yurong,Shi Zhiguang,Zhang Jia,Wang Dongjin,Chen Shaoliang,Wang Liansheng,Gu Aihua,Chen Feng,Yang Tao,Sun Kangyun,Han Yi^ORCID,Xie Liping^ORCID,Chen Hongshan^ORCID,Ji Yong^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

https://www.nature.com/articles/s41422-023-00812-4.pdf

Reference67 articles.

1. Stamler, J. S. Redox signaling: nitrosylation and related target interactions of nitric oxide. Cell 78, 931–936 (1994).

2. Stamler, J. S., Singel, D. J. & Loscalzo, J. Biochemistry of nitric oxide and its redox-activated forms. Science 258, 1898–1902 (1992).

3. Zhao, Y., Vanhoutte, P. M. & Leung, S. W. Vascular nitric oxide: beyond eNOS. J. Pharmacol. Sci. 129, 83–94 (2015).

4. Hess, D. T. et al. Protein S-nitrosylation: purview and parameters. Nat. Rev. Mol. Cell Biol. 6, 150–166 (2005).

5. Lima, B., Forrester, M. T., Hess, D. T. & Stamler, J. S. S-nitrosylation in cardiovascular signaling. Circ. Res. 106, 633–646 (2010).

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