R-Ras is a global regulator of vascular regeneration that suppresses intimal hyperplasia and tumor angiogenesis
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
http://www.nature.com/articles/nm1324.pdf
Reference30 articles.
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2. Cole, A.L., Subbanagounder, G., Mukhopadhyay, S., Berliner, J.A. & Vora, D.K. Oxidized phospholipid-induced endothelial cell/monocyte interaction is mediated by a cAMP-dependent R-Ras/PI3-kinase pathway. Arterioscler. Thromb. Vasc. Biol. 23, 1384–1390 (2003).
3. Ehrhardt, A., Ehrhardt, G.R., Guo, X. & Schrader, J.W. Ras and relatives–job sharing and networking keep an old family together. Exp. Hematol. 30, 1089–1106 (2002).
4. Wang, B., Zou, J.X., Ek-Rylander, B. & Ruoslahti, E. R-Ras contains a proline-rich site that binds to SH3 domains and is required for integrin activation by R-Ras. J. Biol. Chem. 275, 5222–5227 (2000).
5. Zou, J.X. et al. An Eph receptor regulates integrin activity through R-Ras. Proc. Natl Acad. Sci. USA 96, 13813–13818 (1999).
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