Presynaptic GABAergic inhibition regulated by BDNF contributes to neuropathic pain induction-Reference-Cited by-同舟云学术

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Presynaptic GABAergic inhibition regulated by BDNF contributes to neuropathic pain induction

Published:2014-10-30 Issue:1 Volume:5 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Chen Jeremy Tsung-chieh,Guo Da,Campanelli Dario,Frattini Flavia,Mayer Florian,Zhou Luming,Kuner Rohini,Heppenstall Paul A.,Knipper Marlies,Hu Jing

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms6331.pdf

Reference60 articles.

1. Melzack, R. & Wall, P. D. Pain mechanisms: a new theory. Science 150, 971–979 (1965).

2. Zeilhofer, H. U., Wildner, H. & Yévenes, G. E. Fast synaptic inhibition in spinal sensory processing and pain control. Physiol. Rev. 92, 193–235 (2012).

3. Knabl, J. et al. Reversal of pathological pain through specific spinal GABAA receptor subtypes. Nature 451, 330–334 (2008).

4. Munro, G., Erichsen, H. K., Rae, M. G. & Mirza, N. R. A question of balance–positive versus negative allosteric modulation of GABA(A) receptor subtypes as a driver of analgesic efficacy in rat models of inflammatory and neuropathic pain. Neuropharmacology 61, 121–132 (2011).

5. Bonin, R. P. & De Koninck, Y. Restoring ionotropic inhibition as an analgesic strategy. Neurosci. Lett. 557 Pt A, 43–51 (2013).

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