MutT Homolog 1 (MTH1) maintains multiple KRAS-driven pro-malignant pathways-Reference-Cited by-同舟云学术

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MutT Homolog 1 (MTH1) maintains multiple KRAS-driven pro-malignant pathways

Published:2014-07-14 Issue:20 Volume:34 Page:2586-2596
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Patel A,Burton D G A,Halvorsen K,Balkan W,Reiner T,Perez-Stable C,Cohen A,Munoz A,Giribaldi M G,Singh S,Robbins D J,Nguyen D M,Rai P

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2014195.pdf

Reference53 articles.

1. Campbell PM, Der CJ Oncogenic Ras and its role in tumor cell invasion and metastasis. Semin Cancer Biol 2004; 14: 105–114.

2. Rajalingam K, Schreck R, Rapp UR, Albert S Ras oncogenes and their downstream targets. Biochim Biophys Acta 2007; 1773: 1177–1195.

3. Mitsushita J, Lambeth JD, Kamata T The superoxide-generating oxidase Nox1 is functionally required for Ras oncogene transformation. Cancer Res 2004; 64: 3580–3585.

4. Arbiser JL, Petros J, Klafter R, Govindajaran B, McLaughlin ER, Brown LF et al. Reactive oxygen generated by Nox1 triggers the angiogenic switch. Proc Natl Acad Sci USA 2002; 99: 715–720.

5. Weinberg F, Hamanaka R, Wheaton WW, Weinberg S, Joseph J, Lopez M et al. Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity. Proc Natl Acad Sci USA 2010; 107: 8788–8793.

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