Defective maintenance of intracellular Ca2+ homeostasis is linked to increased muscle fatigability in the MG29 null mice-Reference-Cited by-同舟云学术

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Defective maintenance of intracellular Ca2+ homeostasis is linked to increased muscle fatigability in the MG29 null mice

Published:2004-10 Issue:5 Volume:14 Page:373-378
ISSN:1001-0602
Container-title:Cell Research
language:en
Short-container-title:Cell Res

Author:

BROTTO Marco A P,NAGARAJ Ramakrishnan Y,BROTTO Leticia S,TAKESHIMA Hiroshi,MA Jianjie,NOSEK Thomas M

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/7290237.pdf

Reference22 articles.

1. Franzini-Armstrong C, Jorgensen AO . Structure and development of E-C coupling units in skeletal muscle. Annu Rev Physiol 1994; 56:509–34.

2. Rios E, Pizarro G . Voltage sensors and calcium channels of excitation-contraction coupling. NIPS 1988; 3:223–7.

3. Fleischer S, Inui M . Biochemistry and biophysics of excitation-contraction coupling. Annu Rev Biophys Biophys Chem 1989; 18:333–64.

4. Suda N, Franzius D, Fleig A, et al. Ca2+-induced Ca2+ release in Chinese hamster ovary (CHO) cells co-expressing dihydropyridine and ryanodine receptors. J Gen Physiol 1997; 109:619–31.

5. MacKrill JJ . Protein-protein interactions in intracellular Ca2+-release channel function. Biochem J 1999; 337(Pt 3):345–61.

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