IL-22BP is produced by eosinophils in human gut and blocks IL-22 protective actions during colitis-Reference-Cited by-同舟云学术

IL-22BP is produced by eosinophils in human gut and blocks IL-22 protective actions during colitis

Published:2015-09-02 Issue:2 Volume:9 Page:539-549
ISSN:1933-0219
Container-title:Mucosal Immunology
language:en
Short-container-title:Mucosal Immunol

Author:

Martin J C,Bériou G,Heslan M,Bossard C,Jarry A,Abidi A,Hulin P,Ménoret S,Thinard R,Anegon I,Jacqueline C,Lardeux B,Halary F,Renauld J-C,Bourreille A,Josien R

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/mi201583.pdf

Reference49 articles.

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3. Andoh, A. et al. Interleukin-22, a member of the IL-10 subfamily, induces inflammatory responses in colonic subepithelial myofibroblasts. Gastroenterology 129, 969–984 (2005).

4. Brand, S. et al. IL-22 is increased in active Crohn’s disease and promotes proinflammatory gene expression and intestinal epithelial cell migration. Am. J. Physiol. Gastrointest. Liver Physiol. 290, G827–G838 (2006).

5. Schmechel, S. et al. Linking genetic susceptibility to Crohn’s disease with Th17 cell function: IL-22 serum levels are increased in Crohn’s disease and correlate with disease activity and IL23R genotype status. Inflamm. Bowel. Dis. 14, 204–212 (2008).

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