Plasmacytoid dendritic cell-derived IFNα modulates Th17 differentiation during early Bordetella pertussis infection in mice
Author:
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
http://www.nature.com/articles/mi2015101.pdf
Reference50 articles.
1. Higgs, R., Higgins, S.C., Ross, P.J. & Mills, K.H. Immunity to the respiratory pathogen Bordetella pertussis. Mucosal Immunol. 5, 485–500 (2012).
2. Warfel, J.M. & Merkel, T.J. Bordetella pertussis infection induces a mucosal IL-17 response and long-lived Th17 and Th1 immune memory cells in nonhuman primates. Mucosal Immunol. 6, 787–796 (2013).
3. Warfel, J.M., Zimmerman, L.I. & Merkel, T.J. Acellular pertussis vaccines protect against disease but fail to prevent infection and transmission in a nonhuman primate model. Proc. Natl Acad. Sci. USA 111, 787–792 (2014).
4. Fedele, G. et al. Bordetella pertussis commits human dendritic cells to promote a Th1/Th17 response through the activity of adenylate cyclase toxin and MAPK-pathways. PLoS One 5, e8734 (2010).
5. Adkins, I. et al. Bordetella adenylate cyclase toxin differentially modulates toll-like receptor-stimulated activation, migration and T cell stimulatory capacity of dendritic cells. PLoS One 9, e104064 (2014).
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