Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway-Reference-Cited by-同舟云学术

Fructose 1,6-bisphosphate, a high-energy intermediate of glycolysis, attenuates experimental arthritis by activating anti-inflammatory adenosinergic pathway

Published:2015-10-19 Issue:1 Volume:5 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Veras Flávio P.,Peres Raphael S.,Saraiva André L. L.,Pinto Larissa G.,Louzada-Junior Paulo,Cunha Thiago M.,Paschoal Jonas A. R.,Cunha Fernando Q.,Alves-Filho José C.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep15171.pdf

Reference45 articles.

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3. Cronstein, B. N., Naime, D. & Ostad, E. The antiinflammatory mechanism of methotrexate: Increased adenosine release at inflamed sites diminishes leukocyte accumulation in an in vivo model of inflammation. J. Clin. Invest. 92, 2675–2682 (1993).

4. Möttönen, T. et al. Comparison of combination therapy with single-drug therapy in e a rly rheumatoid arthritis : a randomised trial. Lancet 353, 1568–1573 (1999).

5. O’Dell, J. R. et al. Therapies for active rheumatoid arthritis after methotrexate failure. N. Engl. J. Med. 369, 307–18 (2013).

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