Acute cardiovascular protective effects of corticosteroids are mediated by non-transcriptional activation of endothelial nitric oxide synthase-Reference-Cited by-同舟云学术

Acute cardiovascular protective effects of corticosteroids are mediated by non-transcriptional activation of endothelial nitric oxide synthase

Published:2002-05 Issue:5 Volume:8 Page:473-479
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Hafezi-Moghadam Ali,Simoncini Tommaso,Yang Zequan,Limbourg Florian P.,Plumier Jean-Christophe,Rebsamen Michela C.,Hsieh Chung-Ming,Chui Dao-Shan,Thomas Kennard L.,Prorock Alyson J.,Laubach Victor E.,Moskowitz Michael A.,French Brent A.,Ley Klaus,Liao James K.

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm0502-473.pdf

Reference44 articles.

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3. Kirwan, J.R. The effect of glucocorticoids on joint destruction in rheumatoid arthritis. The Arthritis and Rheumatism Council Low-Dose Glucocorticoid Study Group. N. Engl. J. Med. 333, 142–146 (1995).

4. Cronstein, B.N., Kimmel, S.C., Levin, R.I., Martiniuk, F. & Weissmann, G. A mechanism for the antiinflammatory effects of corticosteroids: The glucocorticoid receptor regulates leukocyte adhesion to endothelial cells and expression of endothelial-leuk 1 and intercellular adhesion molecule 1. Proc. Natl. Acad. Sci. USA 89, 9991–9995 (1992).

5. Mangelsdorf, D.J. et al. The nuclear receptor superfamily: The second decade. Cell 83, 835–839 (1995).

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