Runx3d controls the abundance and functional differentiation of CD4+CD8αα+ intraepithelial T cells
Author:
Funder
U.S. Department of Health & Human Services | NIH | National Cancer Institute
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology
Link
https://www.nature.com/articles/s41420-023-01415-z.pdf
Reference14 articles.
1. Zhou C, Qiu Y, Yang H. CD4CD8αα IELs: they have something to say. Front Immunol. 2019;10:2269.
2. Park Y, Moon SJ, Lee SW. Lineage re-commitment of CD4CD8αα intraepithelial lymphocytes in the gut. BMB Rep. 2016;49:11–7.
3. Das G, Augustine MM, Das J, Bottomly K, Ray P, Ray A. An important regulatory role for CD4+CD8 alpha alpha T cells in the intestinal epithelial layer in the prevention of inflammatory bowel disease. Proc Natl Acad Sci USA. 2003;100:5324–9.
4. Li C, Prakhar P, Park JH. The homeostatic gammac cytokines IL-7 and IL-15 suppress the induction of CD4(+)CD8αα(+) intraepithelial T cells in the gut. Cell Mol Immunol. 2022;19:751–3.
5. Li C, Kim HK, Prakhar P, Luo S, Crossman A, Ligons DL, et al. Chemokine receptor CCR9 suppresses the differentiation of CD4(+)CD8αα(+) intraepithelial T cells in the gut. Mucosal Immunol. 2022;15:882–95.
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