Heme oxygenase-1 promoter polymorphisms: do they modulate neonatal hyperbilirubinemia?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Obstetrics and Gynecology,Pediatrics, Perinatology and Child Health
Link
https://www.nature.com/articles/jp20176.pdf
Reference34 articles.
1. Kaplan M, Hammerman C Hereditary contribution to neonatal hyperbilirubinemia. In: Polin RA, Abman SH, Rowitch DH, Benitz WE, Fox WW (eds). Fetal and Neonatal Physiology, 5th edn. Elsevier: Philadelphia, PA, USA, 2017, pp 933–942.
2. Lin Z, Fontaine J, Watchko JF . Coexpression of gene polymorphisms involved in bilirubin production and metabolism. Pediatrics 2008; 122 (1): e156–e162.
3. Tenhunen R, Marver HS, Schmid R . The enzymatic conversion of heme to bilirubin by microsomal heme oxygenase. Proc Natl Acad Sci USA 1968; 61 (2): 748–755.
4. Maines MD, Trakshel GM, Kutty RK . Characterization of two constitutive forms of rat liver microsomal heme oxygenase. Only one molecular species of the enzyme is inducible. J Biol Chem 1986; 261 (1): 411–419.
5. McCoubrey WK Jr, Huang TJ, Maines MD . Isolation and characterization of a cDNA from the rat brain that encodes hemoprotein heme oxygenase-3. Eur J Biochem 1997; 247 (2): 725–732.
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