Preservation of forelimb function by UPF1 gene therapy in a rat model of TDP-43-induced motor paralysis-Reference-Cited by-同舟云学术

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Preservation of forelimb function by UPF1 gene therapy in a rat model of TDP-43-induced motor paralysis

Published:2014-11-06 Issue:1 Volume:22 Page:20-28
ISSN:0969-7128
Container-title:Gene Therapy
language:en
Short-container-title:Gene Ther

Author:

Jackson K L,Dayton R D,Orchard E A,Ju S,Ringe D,Petsko G A,Maquat L E,Klein R L

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,Molecular Medicine

Link

http://www.nature.com/articles/gt2014101.pdf

Reference39 articles.

1. Arai T, Hasegawa M, Akiyama H, Ikeda K, Nonaka T, Mori H et al. TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis. Biochem Biophys Res Commun 2006; 351: 602–611.

2. Mackenzie IR, Bigio EH, Ince PG, Geser F, Neumann M, Cairns NJ et al. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations. Ann Neurol 2007; 61: 427–434.

3. Neumann M, Sampathu DM, Kwong LK, Truax AC, Micsenyi MC, Chou TT et al. Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis. Science 2006; 314: 130–133.

4. Chen-Plotkin AS, Lee VM, Trojanowski JQ . TAR DNA-binding protein 43 in neurodegenerative disease. Nat Rev Neurol 2010; 6: 211–220.

5. Tatom JB, Wang DB, Dayton RD, Skalli O, Hutton ML, Dickson DW et al. Mimicking aspects of frontotemporal lobar degeneration and Lou Gehrig’s disease in rats via TDP-43 overexpression. Mol. Ther. 2009; 17: 607–613.

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