Lack of support for an association between CLEC4M homozygosity and protection against SARS coronavirus infection-Reference-Cited by-同舟云学术

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Lack of support for an association between CLEC4M homozygosity and protection against SARS coronavirus infection

Published:2007-06 Issue:6 Volume:39 Page:691-692
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Tang Nelson Leung-Sang,Chan Paul Kay-Sheung,Hui David Shu-Cheong,To Ka-Fai,Zhang Weimin,Chan Francis K L,Sung Joseph Jao-Yiu,Lo Yuk Ming Dennis

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng0607-691.pdf

Reference7 articles.

1. Chan, V.S. et al. Nat. Genet. 38, 38–46 (2006).

2. Feinberg, H. et al. J. Biol. Chem. 280, 1327–1335 (2005).

3. Snyder, G.A. et al. J. Virol. 79, 4589–4598 (2005).

4. Liu, H. et al. Eur. J. Hum. Genet. 13, 707–715 (2005).

5. Tang, N.L. et al. Clin. Chem. 51, 2333–2340 (2005).

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1. The Role of Host Genetic Factors in Coronavirus Susceptibility: Review of Animal and Systematic Review of Human Literature;2020-06-03

2. Genetic variation in the C-type lectin receptor CLEC4M in type 1 von Willebrand Disease patients;PLOS ONE;2018-02-01

3. Comprehensive assessment showed no associations of variants at the SLC10A1 locus with susceptibility to persistent HBV infection among Southern Chinese;Scientific Reports;2017-04-21

4. Functional polymorphisms of the CCL2 and MBL genes cumulatively increase susceptibility to severe acute respiratory syndrome coronavirus infection;Journal of Infection;2015-07

5. The VNTR polymorphism of the CLEC4M gene and susceptibility to HIV-1 infection in Han Chinese population;Infection, Genetics and Evolution;2013-07

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