Fosl1 is a transcriptional target of c-Fos during osteoclast differentiation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics
Link
http://www.nature.com/articles/ng0200_184.pdf
Reference30 articles.
1. Wang, Z.Q. et al. Bone and haematopoietic defects in mice lacking c-fos. Nature 360, 741–745 ( 1992).
2. Johnson, R.S., Spiegelman, B.M. & Papaioannou, V. Pleiotropic effects of a null mutation in the c- fos proto-oncogene. Cell 71, 577– 586 (1992).
3. Grigoriadis, A.E. et al. c-Fos: a key regulator of osteoclast-macrophage lineage determination and bone remodeling. Science 266, 443– 448 (1994).
4. Wisdon, R. & Verma, I.M. Transformation by Fos proteins requires a C-terminal transactivation domain. Mol. Cell. Biol. 13, 7429–7438 (1993).
5. Jooss, K.U., Funk, M. & Müller, R. An autonomous N-terminal transactivation domain in Fos protein plays a crucial role in transformation. EMBO J. 13, 1467–1475 (1994).
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