Loss of RAGE prevents chronic intermittent hypoxia-induced nonalcoholic fatty liver disease via blockade of NF-кB pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,Molecular Medicine
Link
https://www.nature.com/articles/s41434-022-00351-4.pdf
Reference30 articles.
1. Wang XJ, Malhi H. Nonalcoholic fatty liver disease. Ann Intern Med. 2018;169:ITC65–ITC80.
2. Marjot T, Moolla A, Cobbold JF, Hodson L, Tomlinson JW. Nonalcoholic fatty liver disease in adults: current concepts in etiology, outcomes, and management. Endocr Rev. 2020;41:bnz009.
3. Lemoine M, Serfaty L. Chronic intermittent hypoxia: a breath of fresh air in the understanding of NAFLD pathogenesis. J Hepatol. 2012;56:20–2.
4. Isaza SC, Del Pozo-Maroto E, Dominguez-Alcon L, Elbouayadi L, Gonzalez-Rodriguez A, Garcia-Monzon C. Hypoxia and non-alcoholic fatty liver disease. Front Med. 2020;7:578001.
5. Wu Q, Li S, Li X, Sui Y, Yang Y, Dong L, et al. Inhibition of advanced glycation endproduct formation by lotus seedpod oligomeric procyanidins through RAGE-MAPK Signaling and NF-kappaB activation in high-fat-diet rats. J Agric Food Chem. 2015;63:6989–98.
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