Functional amounts of dystrophin produced by skipping the mutated exon in the mdx dystrophic mouse-Reference-Cited by-同舟云学术

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Functional amounts of dystrophin produced by skipping the mutated exon in the mdx dystrophic mouse

Published:2003-07-06 Issue:8 Volume:9 Page:1009-1014
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Lu Qi Long,Mann Christopher J,Lou Fang,Bou-Gharios George,Morris Glenn E,Xue Shao-an,Fletcher Sue,Partridge Terence A,Wilton Stephen D

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm897.pdf

Reference26 articles.

1. Hoffman, E.P., Brown, R.H. Jr. & Kunkel, L.M. Dystrophin: the protein product of the Duchenne muscular dystrophy locus. Cell 51, 919–928 (1987).

2. Laing, N.G. Molecular genetics and genetic counselling for Duchenne/Becker muscular dystrophy. in Molecular and Cell Biology of Muscular Dystrophy (ed. Partridge, T.) 37–84 (Chapman & Hall, London, 1993).

3. Beauchamp, J.R., Morgan, J.E., Pagel, C.N. & Partridge, T.A. Dynamics of myoblast transplantation reveal a discrete minority of precursors with stem cell-like properties as the myogenic source. J. Cell. Biol. 144, 1113–1122 (1999).

4. Liu, F., Nishikawa, M., Clemens, P.R. & Huang, L. Transfer of full-length Dmd to the diaphragm muscle of Dmd(mdx/mdx) mice through systemic administration of plasmid DNA. Mol. Ther. 4, 45–51 (2001).

5. Yuasa, K. et al. Adeno-associated virus vector-mediated gene transfer into dystrophin-deficient skeletal muscles evokes enhanced immune response against the transgene product. Gene Ther. 9, 1576–1588 (2002).

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