SARS-CoV-2 viral persistence in lung alveolar macrophages is controlled by IFN-γ and NK cells
Author:
Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41435-024-00284-5.pdf
Reference5 articles.
1. Clayton KL, Garcia JV, Clements JE, Walker BD. HIV infection of macrophages: implications for pathogenesis and cure. Pathog Immun. 2017;2:179–92.
2. Brewer RC, Robinson WH, Lanz TV. SARS-CoV-2 infection of monocytes: balancing acts of antibodies and inflammasomes. Signal Transduct Target Ther. 2022;7:1–2.
3. Huot N, Planchais C, Rosenbaum P, Contreras V, Jacquelin B, Petitdemange C, et al. SARS-CoV-2 viral persistence in lung alveolar macrophages is controlled by IFN-γ and NK cells. Nat Immunol. 2023;24:2068–79.
4. Lin L, Spoor MS, Gerth AJ, Brody SL, Peng SL. Modulation of Th1 activation and inflammation by the NF-kappaB repressor Foxj1. Science. 2004;303:1017–20.
5. Huot N, Rascle P, Petitdemange C, Contreras V, Stürzel CM, Baquero E, et al. SIV-induced terminally differentiated adaptive NK cells in lymph nodes associated with enhanced MHC-E restricted activity. Nat Commun. 2021;12:1282.
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