Expression of a noncoding RNA is elevated in Alzheimer's disease and drives rapid feed-forward regulation of β-secretase-Reference-Cited by-同舟云学术

Expression of a noncoding RNA is elevated in Alzheimer's disease and drives rapid feed-forward regulation of β-secretase

Published:2008-06-29 Issue:7 Volume:14 Page:723-730
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Faghihi Mohammad Ali,Modarresi Farzaneh,Khalil Ahmad M,Wood Douglas E,Sahagan Barbara G,Morgan Todd E,Finch Caleb E,St. Laurent III Georges,Kenny Paul J,Wahlestedt Claes

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1784.pdf

Reference50 articles.

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3. Monaco, S., Zanusso, G., Mazzucco, S. & Rizzuto, N. Cerebral amyloidoses: molecular pathways and therapeutic challenges. Curr. Med. Chem. 13, 1903–1913 (2006).

4. Lacor, P.N. et al. Aβ oligomer–induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease. J. Neurosci. 27, 796–807 (2007).

5. Zhu, D. et al. Phospholipases A2 mediate amyloid-β peptide–induced mitochondrial dysfunction. J. Neurosci. 26, 11111–11119 (2006).

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