Cardiac defects and altered ryanodine receptor function in mice lacking FKBP12-Reference-Cited by-同舟云学术

Cardiac defects and altered ryanodine receptor function in mice lacking FKBP12

Published:1998-01 Issue:6666 Volume:391 Page:489-492
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Shou Weinian,Aghdasi Bahman,Armstrong Dawna L.,Guo Qiuxia,Bao Shideng,Charng Min-Ji,Mathews Lawrence M.,Schneider Michael D.,Hamilton Susan L.,Matzuk Martin M.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/35146.pdf

Reference30 articles.

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2. Wang, T.et al. The immunophilin FKBP12 functions as a common inhibitor of the TGFβ family type I receptors. Cell 86, 435–444 (1996).

3. Chen, Y.-G., Liu, F. & Massagué, J. Mechanisms of TGFβ receptor inhibition by FKBP12. EMBO 13, 3866–3876 (1997).

4. Charng, M.-J., Kinnunen, P., Hawker, J., Brand, T. & Schneider, M. D. FKBP12 recognition is dispensable for signal generation by type I transforming growth factor-β receptors. J. Biol. Chem. 271, 22941–22944 (1996).

5. Okadome, T.et al. Characterization of the interaction of FKBP12 with the transforming growth factor-β type 1 receptor in vivo. J. Biol. Chem. 271, 21687–21690 (1996).

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