β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain-Reference-Cited by-同舟云学术

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β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain

Published:2016-08-19 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Chen Gang,Xie Rou-Gang,Gao Yong-Jing,Xu Zhen-Zhong,Zhao Lin-Xia,Bang Sangsu,Berta Temugin,Park Chul-Kyu,Lay Mark,Chen Wei,Ji Ru-Rong

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms12531.pdf

Reference68 articles.

1. Basbaum, A. I., Bautista, D. M., Scherrer, G. & Julius, D. Cellular and molecular mechanisms of pain. Cell 139, 267–284 (2009).

2. Hucho, T. & Levine, J. D. Signaling pathways in sensitization: toward a nociceptor cell biology. Neuron 55, 365–376 (2007).

3. Woolf, C. J. & Salter, M. W. Neuronal plasticity: increasing the gain in pain. Science 288, 1765–1769 (2000).

4. Ji, R. R., Kohno, T., Moore, K. A. & Woolf, C. J. Central sensitization and LTP: do pain and memory share similar mechanisms? Trends Neurosci. 26, 696–705 (2003).

5. Kuner, R. Central mechanisms of pathological pain. Nat. Med. 16, 1258–1266 (2010).

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