Analysis of a polycytosine tract and heteroplasmic length variation in the mitochondrial DNA D-loop of patients with diabetes, MELAS syndrome and race-matched controls-Reference-Cited by-同舟云学术

Analysis of a polycytosine tract and heteroplasmic length variation in the mitochondrial DNA D-loop of patients with diabetes, MELAS syndrome and race-matched controls

Published:2001-05 Issue:5 Volume:18 Page:413-416
ISSN:0742-3071
Container-title:Diabetic Medicine
language:en
Short-container-title:

Author:

Gill-Randall R.,Sherratt E. J.,Thomas A. W.,Gagg J. W.,Lee A.,Alcolado J. C.

Publisher

Wiley

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1464-5491.2001.00477.x/fullpdf

Reference15 articles.

1. Mitochondrial DNA defects: a widening clinical spectrum of disorders;Sherratt;Clin Sci,1997

2. Importance of maternal history of non-insulin dependent diabetic patients;Alcolado;Br Med J,1991

3. Mutation in mitochondrial tRNA.Leu (UUR) gene in a large pedigree with maternally transmitted type II diabetes mellitus and deafness;Van den Ouweland;Nature Genet,1992

4. A mutation in the tRNAleu (UUR) gene associated with the MELAS subgroup of mitochondrial encephalomyopathies;Goto;Nature,1990

5. Mitochondrial diabetes revisited;Ballinger;Nature Genet,1994

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