Genetic inactivation of prohormone convertase (PC1) causes a reduction in cholecystokinin (CCK) levels in the hippocampus, amygdala, pons and medulla in mouse brain that correlates with the degree of colocalization of PC1 and CCK mRNA in these structures
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1471-4159.2003.02295.x/fullpdf
Reference42 articles.
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2. CCK mRNA expression, pro-CCK processing, and regulated secretion of immunoreactive CCK peptides by rat insulinoma (RIN 5F) and mouse pituitary tumor (AtT-20) cells in culture;Beinfeld;Neuropeptides,1992
3. An introduction to neuronal cholecystokinin;Beinfeld;Peptides,2001
4. What we know and what we need to know about the role of endogenous CCK in psychostimulant sensitization;Beinfeld;Life Sci.,2003a
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