The chemokine receptor CCR5 is not a necessary inflammatory mediator in kainic acid-induced hippocampal injury: evidence for a compensatory effect by increased CCR2 and CCR3-Reference-Cited by-同舟云学术

The chemokine receptor CCR5 is not a necessary inflammatory mediator in kainic acid-induced hippocampal injury: evidence for a compensatory effect by increased CCR2 and CCR3

Published:2004-02-04 Issue:1 Volume:86 Page:61-68
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Chen Zhiguo,Yu Shuo,Bakhiet Moiz,Winblad Bengt,Zhu Jie

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1471-4159.2003.01807.x/fullpdf

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3. Distribution, cellular localization and functional role of CCR2 chemokine receptors in adult rat brain;Banisadr;J. Neurochem.,2002

4. CCR5 binds multiple CC-chemokines: MCP-3 acts as a natural antagonist;Blanpain;Blood,1999

5. Differential blood-brain barrier breakdown and leucocyte recruitment following excitotoxic lesions in juvenile and adult rats;Bolton;Exp. Neurol.,1998

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