Free radical-dependent nuclear localization of Bcl-2 in the central nervous system of aged rats is not associated with Bcl-2-mediated protection from apoptosis-Reference-Cited by-同舟云学术

Free radical-dependent nuclear localization of Bcl-2 in the central nervous system of aged rats is not associated with Bcl-2-mediated protection from apoptosis

Published:2004-02-04 Issue:4 Volume:87 Page:981-994
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Kaufmann Joel A.,Perez Mike,Zhang WenRu,Bickford Paula C.,Holmes Douglas B.,Taglialatela Giulio

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1471-4159.2003.02092.x/fullpdf

Reference128 articles.

1. Relationship between susceptibility to protein oxidation, aging, and maximum life span potential of different species;Agarwal;Exp. Gerontol.,1996

2. Multiple subcellular localization of Bcl-2: detection in nuclear outer membrane, endoplasmic reticulum membrane, and mitochondrial membranes;Akao;Cancer Res.,1994

3. Bcl-2 and Bcl-XL serve an anti-inflammatory function in endothelial cells through inhibition of NF-kappaB;Badrichani;J. Clin. Invest.,1999

4. Bcl-2 inhibits p53 nuclear import following DNA damage;Beham;Oncogene,1997

5. Antioxidant-rich diets improve cerebellar physiology and motor learning in aged rats;Bickford;Brain Res.,2000

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