Neurotoxic Aβ peptides increase oxidative stress in vivo through NMDA-receptor and nitric-oxide-synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitro-Reference-Cited by-同舟云学术

Neurotoxic Aβ peptides increase oxidative stress in vivo through NMDA-receptor and nitric-oxide-synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitro

Published:2001-12-20 Issue:4 Volume:76 Page:1050-1056
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Parks Janice K.,Smith Trisha S.,Trimmer Patricia A.,Bennett James P.,Parker W. Davis

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1471-4159.2001.00112.x/fullpdf

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5. Beta-amyloid fragment 25-35 selectively decreases complex IV activity in isolated mitochondria;Canevari;FEBS Lett.,1999

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