Mitochondria deficient in complex I activity are depolarized by hydrogen peroxide in nerve terminals: relevance to Parkinson's disease
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1471-4159.2001.00060.x/fullpdf
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3. Depolarization of in situ mitochondria due to hydrogen peroxide-induced oxidative stress in nerve terminals: inhibition of alpha-ketoglutarate dehydrogenase;Chinopoulos;J. Neurochem.,1999
4. Exacerbated responses to oxidative stress by an Na+ load in isolated nerve terminals: the role of ATP depletion and rise of [Ca2+]i;Chinopoulos;J. Neurosci.,2000
5. Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow;Cohen;Proc. Natl Acad. Sci. USA,1997
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