Impaired proliferation and increased L‐lactate production of dermal fibroblasts in the GK‐rat, a spontaneous model of non‐insulin dependent diabetes mellitus-Reference-Cited by-同舟云学术

Impaired proliferation and increased L‐lactate production of dermal fibroblasts in the GK‐rat, a spontaneous model of non‐insulin dependent diabetes mellitus

Published:1999-01 Issue:1 Volume:7 Page:65-71
ISSN:1067-1927
Container-title:Wound Repair and Regeneration
language:en
Short-container-title:Wound Repair and Regeneration

Author:

Hehenberger Karin¹,Hansson Anders¹,Heilborn Johan D.²,Abdel‐Halim Samy M.¹,Östensson Claes‐Göran¹,Brismar Kerstin¹

Affiliation:

1. From the Department of Molecular Medicine, The Endocrine and Diabetes Unit, and Department of Dermatology and Venereology,

2. Karolinska Institute and Hospital, Stockholm, Sweden

Publisher

Wiley

Subject

Dermatology,Surgery

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1046/j.1524-475x.1999.00065.x

Reference27 articles.

1. Fibroblasts derived from human chronic diabetic wounds have a decreased proliferation rate, which is recovered by the addition of heparin

2. High glucose-induced growth factor resistance in human fibroblasts can be reversed by antioxidants and protein kinase C-inhibitors

3. Phorbol ester‐induced serine phosphorylation of the insulin receptor decreases its tyrosine kinase activity;Takayama S;J Biol Chem,1988

4. Prevention by protein kinase C inhibitors of glucose-induced insulin-receptor tyrosine kinase resistance in rat fat cells

5. Inhibited proliferation of fibroblasts derived from chronic diabetic wounds and normal dermal fibroblasts treated with high glucose is associated with increased formation of L-lactate

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