Rescue of γ2 subunit-deficient mice by transgenic overexpression of the GABAAreceptor γ2S or γ2L subunit isoforms
Author:
Publisher
Wiley
Subject
General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1460-9568.2000.00159.x/fullpdf
Reference22 articles.
1. Rescue of γ2 subunit-deficient mice by transgenic overexpression of the γ2S or γ2L subunit;Baer;Soc. Neurosci. Abstract.,1999a
2. Postsynaptic clustering of GABAA receptors by the γ3 subunit in vivo;Baer;Proc. Natl Acad. Sci. USA,1999b
3. GABAA receptor subtypes differentiated by their γ-subunit variants: prevalence, pharmacology and subunit architecture;Benke;Neuropharmacology,1996
4. Role of brain allopregnanolone in the plasticity of γ-aminobutyric acid type A receptor in rat brain during pregnancy and after delivery;Concas;Proc. Natl Acad. Sci. USA,1998
5. Subcellular localization and endocytosis of homomeric γ2 subunit splice variants of γ-aminobutyric acid type A receptors;Connolly;Mol. Cell. Neurosci.,1999
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