Transient forebrain ischemia induces persistent hyperactivity of large conductance Ca2+-activated potassium channels via oxidation modulation in rat hippocampal CA1 pyramidal neurons
Author:
Publisher
Wiley
Subject
General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1460-9568.2002.01908.x/fullpdf
Reference23 articles.
1. Differential changes of potassium currents in CA1 pyramidal neurons after transient forebrain ischemia;Chi;J. Neurophysiol.,2000
2. Anoxia-induced LTP of isolated NMDA receptor-mediated synaptic responses;Crepel;J. Neurophysiol.,1993a
3. A selective LTP of NMDA receptor-mediated currents induced by anoxia in CA1 hippocampal neurons;Crepel;J. Neurophysiol.,1993b
4. Changes in membrane properties of CA1 pyramidal neurons after transient forebrain ischemia in vivo;Gao;Neuroscience,1999
5. Enhancement in activities of large conductance calcium-activated potassium channels in CA1 pyramidal neurons of rat hippocampus after transient forebrain ischemia;Gong;Brain Res.,2000
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